‘Unraveling the Key to a Cold Virus’s Effectiveness’

Carl Zimmer, writing for the New York Times:

At body temperature, the cells responded with a sophisticated defense, sending out warning signals to uninfected cells around them. Those cells prepared an arsenal of antiviral proteins, which they used to destroy the rhinoviruses.

But at a relatively cool 91.4 degrees Fahrenheit, Dr. Iwasaki and her colleagues found, things changed.

The neighboring cells only managed a weak defense, allowing the rhinoviruses to invade them and multiply. This result pointed to an explanation for why rhinoviruses plague humans at low temperatures: In cool conditions, the immune system somehow falters.

Next time I feel a cold coming on, I’ll crank the thermostat up to 98.6 °F.

Further updates to follow.